anti-E. coli O157 LPS Antibody

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Catalog number: AM31451PU-N
Price: 1036 €
Supplier: acr
Product name: anti-E. coli O157 LPS Antibody
Quantity: 1 mg
Other quantities: 1 mg 1036€
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More details :

Vial description: EHEC, EPEC, ETEC, Escherichia coli
Category: primary antibody
Clonality: monoclonal
Raised in: Mouse
Clone: B839M
Form: Purified
Reactivity: not determined
Purity: purified
Molecular weight: in kDa
Source: mammalian
Activity: to be determined
Range: broad
Protein number: see ncbi
Application: Immunocytochemistry/Immunofluorescence, ELISA
Storage: Avoid freeze/thaw cycles, Upon delivery aliquot and store at -20°, C, C for one year, Shipped at 4°
Description: Freeze thaw will destroy a percentage in every cycle and should be avoided, C in a fridge short term in a concentrated dilution, This antibody needs to be stored at + 4°
Properties: C, C for long term storage and for short term at + 5°, If you buy Antibodies supplied by acr they should be stored frozen at - 24°
Gene: According to the current model, Agonistic forms of LPS or lipid A trigger numerous physiological immunostimulatory effects in mammalian organisms, Cells of the myeloid lineage have been shown to be the primary cellular sensors for LPS in the mammalian immune system, During the past decade, In addition, LPS consist of a poly- or oligosaccharide region that is anchored in the outer bacterial membrane by a specific carbohydrate lipid moiety termed lipid A, Some members of the latter more heterogeneous group are capable of antagonizing the effects of strongly stimulatory LPS/lipid A forms, The elucidation of structure-activity correlations in LPS and lipid A has not only contributed to a molecular understanding of both immunostimulatory and toxic septic processes, The lipid A component is the primary immunostimulatory center of LPS, With respect to immunoactivation in mammalian systems, but has also re-animated the development of new pharmacological and immuno-stimulatory strategies for the prevention and therapy of infectious and malignant diseases, but--in higher doses--can also lead to pathological reactions such as the induction of septic shock, enormous progress has been obtained in the elucidation of the central LPS/lipid A recognition and signaling system in mammalian phagocytes, leading to the rapid activation of an intracellular signaling network that is highly homologous to the signaling systems of IL-1 and IL-18, several natural or derivative lipid A structures have been identified that display comparatively low or even no immunostimulation for a given mammalian species, the classical group of strongly agonistic (highly endotoxin) forms of LPS has been shown to be comprised of a rather similar set of lipid A types, the membrane-bound or soluble forms of CD14 and the newly identified Toll-like receptor 4 (TLR4)*MD-2 complex, the specific cellular recognition of agonistic LPS/lipid A is initialized by the combined extracellular actions of LPS binding protein (LBP), Bacterial pathogen lipopolysaccharides (LPS) are the major outer surface membrane components present in almost all Gram-negative bacteria and act as extremely strong stimulators of innate or natural immunity in diverse eukaryotic species ranging from insects to humans | More about : According to the current model, Agonistic forms of LPS or lipid A trigger numerous physiological immunostimulatory effects in mammalian organisms, Cells of the myeloid lineage have been shown to be the primary cellular sensors for LPS in the mammalian immune system, During the past decade, In addition, LPS consist of a poly- or oligosaccharide region that is anchored in the outer bacterial membrane by a specific carbohydrate lipid moiety termed lipid A, Some members of the latter more heterogeneous group are capable of antagonizing the effects of strongly stimulatory LPS/lipid A forms, The elucidation of structure-activity correlations in LPS and lipid A has not only contributed to a molecular understanding of both immunostimulatory and toxic septic processes, The lipid A component is the primary immunostimulatory center of LPS, With respect to immunoactivation in mammalian systems, but has also re-animated the development of new pharmacological and immuno-stimulatory strategies for the prevention and therapy of infectious and malignant diseases, but--in higher doses--can also lead to pathological reactions such as the induction of septic shock, enormous progress has been obtained in the elucidation of the central LPS/lipid A recognition and signaling system in mammalian phagocytes, leading to the rapid activation of an intracellular signaling network that is highly homologous to the signaling systems of IL-1 and IL-18, several natural or derivative lipid A structures have been identified that display comparatively low or even no immunostimulation for a given mammalian species, the classical group of strongly agonistic (highly endotoxin) forms of LPS has been shown to be comprised of a rather similar set of lipid A types, the membrane-bound or soluble forms of CD14 and the newly identified Toll-like receptor 4 (TLR4)*MD-2 complex, the specific cellular recognition of agonistic LPS/lipid A is initialized by the combined extracellular actions of LPS binding protein (LBP), Bacterial pathogen lipopolysaccharides (LPS) are the major outer surface membrane components present in almost all Gram-negative bacteria and act as extremely strong stimulators of innate or natural immunity in diverse eukaryotic species ranging from insects to humans
Gene target: O157 LPS
Short name: coli O157 LPS Antibody, anti-E
Technique: antibodies against human proteins, antibodies for, antibody to, Antibody
Host: Escherichia coli
Species: E, coli, coli, E
Alternative name: coli O157 LPS (Antibody to), antibody to-E
Alternative technique: antibodies

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